Artículo

Baculovirus-based gene silencing of Humanin for the treatment of pituitary tumors

Gottardo, María Florencia Icon; Pidre, Matias Luis Icon; Zuccato, Camila Florencia Icon; Asad, Antonela Sofía Icon; Imsen, Mercedes Icon; Jaita, Gabriela Alejandra Icon; Candolfi, Marianela Icon; Romanowski, Victor Icon; Seilicovich, Adriana Icon

Otros responsables

Fecha

02/2018

Editorial

Springer

Revista

Apoptosis

Título del libro

Nombre del evento

Fecha del evento

Institución organizadora

ISSN

1360-8185

eISSN

ISBN

Idioma

Inglés

Tipo del evento

Resumen

Pituitary tumors are the most common primary intracranial neoplasms. Humanin (HN) and Rattin (HNr), a rat homolog of HN, are short peptides with a cytoprotective action. In the present study, we aimed to evaluate whether endogenous HNr plays an antiapoptotic role in pituitary tumor cells. Thus, we used RNA interference based on short-hairpin RNA (shRNA) targeted to HNr (shHNr). A plasmid including the coding sequences for shHNr and dTomato fluorescent reporter gene was developed (pUC-shHNr). Transfection of somatolactotrope GH3 cells with pUC-shHNr increased apoptosis, suggesting that endogenous HNr plays a cytoprotective role in pituitary tumor cells. In order to evaluate the effect of blockade of endogenous HNr expression in vivo, we constructed a recombinant baculovirus (BV) encoding shHNr (BV-shHNr). In vitro, BV-shRNA was capable of transducing more than 80% of GH3 cells and decreased HNr mRNA. Also, BV-shHNr increased apoptosis in transduced GH3 cells. Intratumor injection of BV-shHNr to nude mice bearing s.c. GH3 tumors increased the number of apoptotic cells, delayed tumor growth and enhanced survival rate, suggesting that endogenous HNr may be involved in pituitary tumor progression. These preclinical data suggests that the silencing of HN expression could have a therapeutic impact on the treatment of pituitary tumors.

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