Artículo

Impaired insulin signaling pathway in ovarian follicles of cows with cystic ovarian disease

Hein, Gustavo Juan Icon; Panzani, Carolina ; Rodríguez, Fernanda Mariel Icon; Salvetti, Natalia Raquel Icon; Díaz, Pablo Uriel Icon; Gareis, Natalia Carolina Icon; Benitez, G. A. ; Ortega, Hugo Hector Icon; Rey, Florencia Icon

Otros responsables

Fecha

05/2015

Editorial

Elsevier Science

Revista

Animal Reproduction Science

Título del libro

Nombre del evento

Fecha del evento

Institución organizadora

ISSN

0378-4320

eISSN

ISBN

Idioma

Inglés

Tipo del evento

Clasificación temática

Resumen

Cystic ovarian disease (COD) is an important cause of infertility in dairy cattle. Follicular cell steroidogenesis and proliferation in ovulatory follicles is stimulated by hormones such as insulin and its necessary post-receptor response. The aim of this study was to determine the expression of insulin receptor (IR), IR substrate-1 (IRS1) and phosphatidylinositol 3-kinase (PI3K), key intermediates in the insulin pathway, in control cows and cows with spontaneous COD and ACTH-induced COD. IR and IRS1 mRNA levels were greater in granulosa cells and lower in follicular cysts than in control tertiary follicles. PI3K mRNA levels were similar in all follicles evaluated, whereas the expression of IR, IRS1 and PI3K was similar in theca cells. Protein expression of IR was higher in control tertiary follicles than in the same structures in animals with COD and with cysts. IRS1 and PI3K protein expression showed the same pattern in tertiary and cystic follicles. However, the protein expression of subunit alpha p85 of PI3K was greater in theca cells from tertiary follicles than in cystic follicles. These results provide new insights into the insulin response in cows with COD. The lower gene and protein expressions of some insulin downstream effectors at an early stage of the signaling pathway could negatively influence the functionality of ovaries and contribute to follicle persistence.

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