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dc.contributor.author
Curciarello, Renata  
dc.contributor.author
Sobande, Toni  
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Jones, Samantha  
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Giuffrida, Paolo  
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Di Sabatino, Antonio  
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Docena, Guillermo H.  
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Macdonald, Thomas T.  
dc.contributor.author
Klaartje, Kok  
dc.date.available
2022-04-08T18:18:26Z  
dc.date.issued
2020-05-22  
dc.identifier.citation
Curciarello, Renata; Sobande, Toni; Jones, Samantha; Giuffrida, Paolo; Di Sabatino, Antonio; et al.; Human neutrophil elastase proteolytic activity in Ulcerative colitis favors the loss of function of therapeutic monoclonal antibodies; Dove Press; Journal of Inflammation Research; 13; 22-5-2020; 233-243  
dc.identifier.issn
1178-7031  
dc.identifier.uri
http://hdl.handle.net/11336/154829  
dc.description.abstract
Purpose: Proteases play an essential role in the pathophysiology of inflammatory bowel disease (IBD), contributing to the intestinal mucosal lesions through the degradation of the extracellular matrix and alteration of the barrier function. Ulcerative colitis (UC) is characterized by an extensive infiltrate of neutrophils into the mucosa and hence, increased proteolytic activity. Human neutrophil elastase (HNE) is a serine protease that has been reported to be increased in UC patients’ intestinal mucosa. Based on our previous studies, we hypothesized that HNE might induce proteolytic degradation and loss of function of therapeutic monoclonal antibodies in IBD patients. Patients and Methods: Elastase expression and elastinolytic activity were determined in mucosal explants from ulcerative colitis patients (n=6) and cultured ex vivo in the presence or absence of recombinant elafin. Enzymatic digestions of therapeutic monoclonal antibodies were performed using recombinant HNE and elafin. The integrity of the therapeutic antibodies was evaluated by immunoblotting and protein G binding assay, whereas their TNFneutralizing activity was assessed with a reporter cell line. Results: We found that HNE and its elastinolytic activity were increased in the gut mucosa of UC patients. We also demonstrated that HNE cleaved biological drugs, impairing the TNF-α neutralizing capacity of anti-TNF monoclonal antibodies. This proteolytic degradation was inhibited by the addition of the specific inhibitor, elafin. Conclusion: Our results suggest that the high level of proteolytic degradation by mucosal neutrophil elastase, along with a potential imbalance with elafin, contributes to the loss of function of biologic agents, which are currently used in patients with IBD. These findings might explain the non-responsiveness of UC patients to therapeutic monoclonal antibodies and suggest the potential beneficial concomitant use of elafin in this treatment.  
dc.format
application/pdf  
dc.language.iso
eng  
dc.publisher
Dove Press  
dc.rights
info:eu-repo/semantics/openAccess  
dc.rights.uri
https://creativecommons.org/licenses/by-nc/2.5/ar/  
dc.subject
ELASTINOLYTIC ACTIVITY  
dc.subject
ELAFIN  
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ANTI-TNF  
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INFLAMMATORY BOWEL DISEASE  
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BIOLOGICAL DRUGS  
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Inmunología  
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Medicina Básica  
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CIENCIAS MÉDICAS Y DE LA SALUD  
dc.title
Human neutrophil elastase proteolytic activity in Ulcerative colitis favors the loss of function of therapeutic monoclonal antibodies  
dc.type
info:eu-repo/semantics/article  
dc.type
info:ar-repo/semantics/artículo  
dc.type
info:eu-repo/semantics/publishedVersion  
dc.date.updated
2021-09-06T17:49:34Z  
dc.journal.volume
13  
dc.journal.pagination
233-243  
dc.journal.pais
Nueva Zelanda  
dc.journal.ciudad
Aukland  
dc.description.fil
Fil: Curciarello, Renata. Consejo Nacional de Investigaciones Científicas y Técnicas. Centro Científico Tecnológico Conicet - La Plata. Instituto de Estudios Inmunológicos y Fisiopatológicos. Universidad Nacional de La Plata. Facultad de Ciencias Exactas. Instituto de Estudios Inmunológicos y Fisiopatológicos; Argentina. University of London; Reino Unido  
dc.description.fil
Fil: Sobande, Toni. University of London; Reino Unido  
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Fil: Jones, Samantha. University of London; Reino Unido  
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Fil: Giuffrida, Paolo. University of London; Reino Unido. Universita Degli Studi Di Pavia; Italia  
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Fil: Di Sabatino, Antonio. Universita Degli Studi Di Pavia; Italia  
dc.description.fil
Fil: Docena, Guillermo H.. Consejo Nacional de Investigaciones Científicas y Técnicas. Centro Científico Tecnológico Conicet - La Plata. Instituto de Estudios Inmunológicos y Fisiopatológicos. Universidad Nacional de La Plata. Facultad de Ciencias Exactas. Instituto de Estudios Inmunológicos y Fisiopatológicos; Argentina  
dc.description.fil
Fil: Macdonald, Thomas T.. University of London; Reino Unido  
dc.description.fil
Fil: Klaartje, Kok. University of London; Reino Unido. Royal London Hospital; Reino Unido  
dc.journal.title
Journal of Inflammation Research  
dc.relation.alternativeid
info:eu-repo/semantics/altIdentifier/url/https://www.dovepress.com/articles.php?article_id=54030  
dc.relation.alternativeid
info:eu-repo/semantics/altIdentifier/doi/http://dx.doi.org/10.2147/JIR.S234710